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CGRP represents the most intriguing biological mechanism, linking the nervous system to bone repair, but direct therapeutic application is complicated by its role in pain signaling and the current clinical use of CGRP-blocking agents for migraine treatment

Sample analysis The method was applied to A549 cells incubated in duplicate with CdCl (50 and 100 mol/L) or cisplatin (100 and 200 mol/L) for 24 and 48 h, alongside untreated controls

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